The gallbladder is a 3-4 inch pear-shaped organ located on the right side of the body, directly under the liver.
The primary function of the gallbladder is to store and release bile
Bile contains cholesterol, bile salts, lecithin and other substances. The bile emulsifies (disperses into smaller globules) fats so that they may be digested. One of the functions of the liver is to remove poisonous substances from the blood so that they can be expelled from the body. The liver excretes all these gathered toxins which are then mixed with bile.
The liver produces about 1 3/4 pints of bile everyday. It goes to the gallbladder first where it is held until food arrives to the small intestine. The gallbladder then releases bile which passes through the cystic and bile ducts into the small intestine. Ultimately the toxins are then passed out of the body.
Abnormal concentration of bile acids, cholesterol and phospholipids in the bile can cause the formation of gallstones. When a gallstone is pushed out of the gallbladder it can lodge in the bile duct and cause nausea, vomiting and pain in the upper right abdominal region. These symptoms can often arise after a person has eaten fatty foods. Gallstones can range from the size of a grain of sand to larger than a pea. 75% of gallstones are cholesterol while the remaining 25% are pigment stones. Pigment stones are comprised of calcium salts.
The presence of gallstones may indicate that gallbladder disease is present. Symptoms may include light colored feces that float, constant pain below the breastbone that shoots up into the right or left shoulder area and transfers to the back. It can be accompanied by fever, nausea and vomiting. Attacks often occur in the evening and can last from 30 minutes to several hours. The urine may be tea or coffee colored and one may experience shaking and chills. Sometimes, a gallbladder attack may mimic a heart attack. Seek treatment for severe pain that does not go away.
Anyone that has had their gallbladder removed should be particularly diligent about improving their diet.
Removing an organ does not improve health. It may reduce symptoms but it does not cure the underlying cause that brought about the symptoms in the first place.
Those that must have their gallbladder removed are sick and unless the conditions that brought about the disease in the first place are not changed, they are still sick after the gallbladder and pain are gone. Other symptoms may present elsewhere later. Gallbladder disease can be the result of a deficiency of neutralizing minerals in the diet. These are primarily found in vegetables and fruits. One of the functions of the gallbladder is to reclaim sodium from the liver bile. Without a gallbladder the ability to recycle sodium is lost reducing the available sodium even more. Unless those who have had their gallbladder removed add enough alkalizing foods in the form of vegetables and fruits to their diets, their bodies are still in danger of becoming too acidic. The problem has not gone away.
Unless the minerals are supplied by the diet, the body will find another source to carry out the function previously supplied by the sodium in the gallbladder. Calcium is the most readily available substitute for sodium and the best source of calcium is the bones. This process can lead to osteoporosis.
– Red Beet Root
– Ginger Root
– Oregon Grape
Supporting the Gallbladder
Foods to consider adding to your diet for supporting gallbladder function are:
– Vegetables and fruits
Eat these foods in moderation:
– Dairy and Meats
If you have severe symptoms, omit these foods entirely as they may contribute to further suppressing the gallbladder function and flaring symtoms:
– Fried and Fatty foods.
Often there is more than one issue going on in the body at any one time. Everything is connected and sometimes the body needs to work in a prioritizing sequence. Don’t be alarmed if multiple supplements are suggested by your healthcare professional. It can be very beneficial to support several different systems and organs of the body as we assist it to heal.
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